Our most well-respected scientific research validates the time-honored understanding that by far and away the most powerful factors affecting health and longevity are modifiable choices including the foods we eat and other aspects of lifestyle. So let’s move forward and explore this empowering information.
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CSF antigliadin antibodies and the Ramsay Hunt syndrome
Neurology • May 2, 1997
Insulin regulation of human ovarian androgens
Human Reproduction Volume 12 • January 1, 1997
Effects of inulin and lactose on fecal microflora, microbial activity, and bowel habit in elderly constipated persons
American Journal of Clinical Nutrition • December 19, 1996
The effect on memory of chronic prednisone treatment in patients with systemic disease
Neurology • December 1, 1996
Essential fatty acid metabolism in boys with attention-deficit hyperactivity disorder
American Journal of Clinical Nutrition • October 1, 1995
Adult celiac disease is frequently associated with sacroiliitis.
Digestive Diseases and Sciences • September 4, 1995
Advanced glycation end products contribute to amyloidosis in Alzheimer disease
Proceedings of the National Academy of Sciences • May 1, 1994
Usefulness of coenzyme Q10 in clinical cardiology: a long-term study.
Moleculaar aspects of Medicine • January 1, 1994
Pathobiology of magnesium deficiency: a cytokine/neurogenic inflammation hypothesis.
American Journal of Physiology • September 19, 1992
Celiac disease, brain atrophy, and dementia
American Academy of Neurology • March 14, 1991
The Contribution of Dental Amalgam to Mercury in Blood
Journal of Dental Research • May 1, 1989
The hippocampus is one of the most affected areas in Alzheimer’s disease (AD)1. Moreover, this structure hosts one of the most unique phenomena of the adult mammalian brain, namely, the addition of new neurons throughout life2. This process, called adult hippocampal neurogenesis (AHN), confers an unparalleled degree of plasticity to the entire hip- pocampal circuitry3,4. Nonetheless, direct evidence of AHN in humans has remained elusive. Thus, determining whether new neurons are continuously incorporated into the human dentate gyrus (DG) during physiological and pathologi- cal aging is a crucial question with outstanding therapeutic potential. By combining human brain samples obtained under tightly controlled conditions and state-of-the-art tissue pro- cessing methods, we identified thousands of immature neu- rons in the DG of neurologically healthy human subjects up to the ninth decade of life. These neurons exhibited variable degrees of maturation along differentiation stages of AHN. In sharp contrast, the number and maturation of these neurons progressively declined as AD advanced. These results dem- onstrate the persistence of AHN during both physiological and pathological aging in humans and provide evidence for impaired neurogenesis as a potentially relevant mechanism underlying memory deficits in AD that might be amenable to novel therapeutic strategies
Anticholinergic drugs and risk of dementia: case-control study
British Medical Journal •
The Empowering Neurologist Podcast
The Empowering Neurologist is my interview series with some of the most exciting practitioners and thought leaders in health and wellness. Each episode features an in-depth discussion about cutting-edge advancements in areas like Alzheimer’s prevention, nutrition, aging, and more.